Pulmonary Pathology by Nagarjun Rao and Cesar A. Moran
Oral and pharyngeal bolus transit in patients with chronic obstructive pulmonary disease Rachel Aguiar Cassiani, Carla Manfredi Santos, José Baddini-Martinez, Roberto Oliveira Dantas Department of Medicine, Medical School of Ribeirão Preto, University of São Paulo, Ribeirão Preto, São Paulo, Brazil Background: Patients with respiratory diseases, such as chronic obstructive pulmonary disease (COPD), may have swallowing dysfunction. Objective: The aim of this investigation was to evaluate the oral and pharyngeal phases of swallowing in patients with COPD. Methods: We studied 16 patients with clinical manifestations and pulmonary function tests diagnosis of COPD (mean age: 68 years) and 15 nonsmoking healthy volunteers (mean age: 65 years) with normal pulmonary function tests. All subjects were submitted to clinical and videofluoroscopic evaluation of swallowing. Each subject performed in duplicate swallows of 5 mL and 10 mL of liquid bolus, paste bolus, and a solid bolus. Results: In general, the duration of the events of the pharyngeal phase of swallowing was longer in COPD patients than controls. The difference was significant in the laryngeal vestibular closure, hyoid movement, and pharyngeal transit with swallows of both volumes of liquid bolus; in oral–pharyngeal transit with 5 mL paste bolus; and in pharyngeal and oral–pharyngeal transit with solid bolus. The difference between the duration of maximal laryngeal elevation and the duration of pharyngeal transit was higher in control subjects than in patients with COPD. Conclusion: The results suggested that patients with COPD have a longer pharyngeal swallowing phase than normal subjects, which is associated with a decrease in the difference between the duration of maximal laryngeal elevation and the duration of pharyngeal transit. Keywords: COPD, videofluoroscopy, oral–pharyngeal transit, swallowing, deglutition
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Download this Hand Drawn Cartoon Chronic Obstructive Pulmonary Disease, Copd, Disease, Pulmonary Disease PNG clipart image with transparent background for free. Pngtree provides millions of free png, vectors, clipart images and psd graphic resources for designers.| 5933706
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Miliary tuberculosis is an uncommon pulmonary manifestation of tuberculosis. Pathology Miliary tuberculosis represents hematogenous dissemination of uncontrolled tuberculous infection and carries a relatively poor prognosis. It is seen in prima...
Can you answer today’s question 🙋♀️?! Q: If a patient aspired something into his lungs while lying down on his back, to which pulmonary segment will it go?! 🤔 …#anatomy #physiology #pathology #biology #lung #medical #surgery #medicine #usmle #nclex #neet
Pathology: A Movie About Snowboarding and Humans. Bryan Fox and Austin Smith partnered with storyteller Liam Gallagher to document a season of their experiences…
Growth Hormone Defects & ExcessPhysiology ReviewSecretion: Growth hormone regulates growth and metabolism in most body tissues. Growth hormone releasing hormone (GHRH) is secreted from the hypothalamus, and travels via neurons to the hypothalamic-pituitary portal system to reach somatotrophs in the anterior pituitary. In response to GHRH, the somatotrophs release growth hormone into the blood. Effects: Growth hormone acts directly on the tissues to increase blood glucose and free fatty acids, and promotes tissue growth and differentiation. Furthermore, growth hormone triggers hepatic release of IGF-1, which enhances the effects of growth hormone. Growth Hormone DefectsGrowth Hormone DeficiencyGrowth hormone deficiency results in reduced levels of growth hormone and, consequently, reduced levels of IGF-1. In most cases, isolated growth hormone deficiency is due to defects in the GH1 gene, which is responsible for growth hormone production.However, we need to rule out general hypopituitarism, in which other anterior pituitary hormones are also low.Treatment: Recombinant human growth hormone. Growth Hormone Insensitivity Growth hormone insensitivity is characterized by normal or high levels of growth hormone, and low levels of IGF-1. Insensitivity is caused by growth hormone receptor defects, which may be hereditary, as in Laron Syndrome, or acquired as a result of malnutrition, liver disease, diabetes, growth hormone receptor antibodies, or other pathologies.Treatment: recombinant IGF-1. Effects of Growth Hormone Defects:In Childhood: - If the defect appears in childhood, linear growth is slow (sometimes called pituitary dwarfism); - These patients often have delayed puberty, with small gonads and genitalia (micropenis).In Adulthood: - If growth hormone defects appear later in life, adults present with body composition changes, including reduced muscle and bone mass, and increased fat mass. - These patients have increased risk of cardiovascular disease (including left...
Orthodox Pathology - ORTHOGNOSIA
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Pathology Secrets: 9780323055949: Medicine & Health Science Books @ Amazon.com
Renal Pathology Hey there everyone, today we're going to go on a journey, a sonographic journey through renal diseases reliably discovered via ultrasound.
Plant Pathology: Agrios, George N: 9780120445653: Books - Amazon.ca
Classification of Bone pathology:- Regards:- Kriti Naja Jain :) Reference :- Neville oral and maxillofacial pathology 3rd e
