Host-Pathogen Interactions in Streptococcal Diseases Taschenbuch von G Singh Chhatwal Details Autor: G. Singh Chhatwal Redaktion: G Singh Chhatwal Herausgeber: G Singh Chhatwal EAN: 9783642438363 Einband: Kartoniert / Broschiert Inhalt: viii / 255 S. Ausstattung / Beilage: Paperback Auflage: 2013 Sprache: Englisch Seiten: 264 Reihe: Current Topics in Microbiology and Immunology Maße: 235 x 155 x 15 mm Erschienen: 12.04.2015 Schlagworte: Biologie / Allgemeines / Lexika / Medizinische Forschung / Mikrobiologie / Public Health und Präventivmedizin / Immunologie / Mikrobiologie und Virologie / Pharmakologie / nicht-medizinisch / Verstehen Beschreibung Streptococci are Gram-positive bacteria that cause a wide spectrum of diseases, such as pharyngitis, necrotizing fasciitis and streptococcal toxic shock syndrome, as well as rheumatic fever and rheumatic heart disease as sequelae. Antibiotics alone have not been able to control the disease and in spite of many efforts an effective vaccine is not yet available. A pre
Host-pathogen interface - Infogalactic: the planetary knowledge core
The observed fusion of membrane vesicles liberated from virulent organisms with closely interacting macrophages is proposed here to constitute a mechanism for type ill secretion-like system of gram-negative organisms in general, in order to translocate the necessary biochemical signals into the cytosol of macrophage (eucaryotic host cells). Ultrastructural studies of experimentally-infected chicken ileum reveal that virulent Salmonellae (3, 10: r: -) signal their own phagocytosis by tissue macrophages, and are thus able to dodge host defence mechanisms. The observed fusion of membrane vesicles (MVs) liberated from virulent organisms with closely interacting macrophages is proposed here to constitute a mechanism for type ill secretion-like system of gram-negative organisms in general, in order to translocate the necessary biochemical signals into the cytosol of macrophages (eucaryotic host cells). This leads to (i) membrane ruftling culminating in augmented phagocytosis of salmonellae, (ii)inhibition of phagosome-lysosome (P-L) fusion for survival and replication of the pathogens within thehost cytoplasm and (iii) initiation of apoptosis of the infected macrophages and ultimate release of salmonellae from the migrating host cells.
Article: A structural model for virulence organellae of gram-negative organisms with reference to Salmonella pathogenicity in chicken ileum
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