There is no set diet for erosive esophagitis, but avoiding caffeine and adopting a bland diet can help symptoms. Talk to a doctor about what to eat and drink.
Learn about esophageal stricture. What are causes & symptoms of esophageal stricture. How is esophageal stricture diagnosed & treated
After researching Eosinophilic Esophagitis, I was aware of the Elimination Diet, but wanted an official diagnosis before putting my big-appetite eater through the toils of a more limited diet.
Esophageal thrush is a fungal infection of the food pipe. It occurs when a naturally occurring fungus called Candida grows out of control. Esophageal thrush is a serious condition, causing pain and difficulty swallowing. It may also spread to other parts of the body. Learn more about esophageal thrush here.
Eosinophilic esophagitis, or EoE, is a chronic, immune-mediated condition where your esophagus develops inflammation. Let's cover EoE basics!
Many people find that a backup of stomach acids irritates their esophagus after eating. Here’s how to stop that bothersome acid reflux and heal your esophagus.
Having trouble swallowing? Learn more about what causes this common issue, along with therapies for treating the condition.
Eosinophilic esophagitis, or EoE, is a chronic, immune-mediated condition where your esophagus develops inflammation. Let's cover EoE basics!
Do you get a burning in your throat or chest after you eat? It could be heartburn -- or over a dozen other conditions.
Learn about eosinophilic esophagitis (EoE) and its symptoms, including chronic cough, treatment options, and diagnosis methods.
Esophageal cancer is when a malignant tumor forms in the lining of the muscular tube through which food moves from the throat to the stomach.
Are you experiencing heartburn and trouble swallowing? It could be esophagitis. Discover causes, symptoms & treatment options.
"If you are struggling with physical pain, no matter how internal and invisible it seems, see a specialist."
ESOPHAGEAL VARICES Esophageal varices develop in response to an increase in venous pressure in a location distal to the azygos vein and the right ventricle. The impedance to flow may be functional, as in a hyperdynamic circulatory state, or mechanical, as with a blood clot or tumor. Further vasodilation of the splanchnic venous system may also result from secondary changes in vascular circulatory mediators such as nitric oxide and vasoactive intestinal peptide. A variety of disorders, ranging from splenic, portal, or hepatic vein thrombosis (BuddChiari syndrome) to rightsided heart failure, may lead to esophageal varices. The most common cause of esophageal varices is portal hypertension secondary to intrahepatic causes, such as cirrhosis. In cirrhosis, there is fibrosis of the sinusoids and shunting that leads to portal vein backflow. As many as half of patients presenting with a new diagnosis of hepatic cirrhosis have esophageal varices on initial evaluation. The vast majority of patients with cirrhosis will also develop esophageal varices over the course of the disease if they do not undergo liver transplantation. If varices are present, there is a greater risk for bleeding with risk factors such as larger varices, increased portal hypertension, hepatic failure, and endoscopic signs of recent or impending bleeding (e.g., red wale signs). Bleeding from esophageal varices may be brisk and massive, and the risk of death is considerable. Many treatments are available for esophageal varices and are used on the basis of whether they are needed for management of acute bleeding, prevention of recurrent bleeding. or prophylaxis in patients with diagnosed nonbleeding varices. Amongst these clinical scenarios, treatments are divided into the broad categories of variceal obliteration (endoscopic banding and sclerosis), pharmacologic reduction of portal venous pressure (betaantagonists, nitrates, somatostatin), and mechanical reduction of portal venous pressure (transhepatic intravascular portosystemic shunt [TIPS], surgical portacaval shunt, liver transplantation). Acute and chronic treatments may be a combination of obliterative and pharmacologic treatments. For example, acute bleeding may be managed by esophageal variceal banding and intravenous octreotide, whereas chronic prevention may rely on banding and use of betaantagonists. Prophylactic therapy tends to be pharmacologic, but obliterative techniques may be used in addition. Therapies such as TIPS and transplant are reserved for more severe and/or refractory cases of variceal bleeding.
Learn more about what Eosinophilic Esophagitis is and how to test and treat it.
Eosinophilic esophagitis, or EoE, is a chronic, immune-mediated condition where your esophagus develops inflammation. Let's cover EoE basics!
Both physicians and people with erosive esophagitis share some effective tips for preventing acid reflux (without having to run to the doctor).
We can’t always control the factors that cause our lower esophageal sphincter to deteriorate, but there are some things we can do to make sure we are caring for our LES, particularly if we fall under the risk factors for GERD. Here’s everything you should know about the lower esophageal sph
There is no set diet for erosive esophagitis, but avoiding caffeine and adopting a bland diet can help symptoms. Talk to a doctor about what to eat and drink.
Dietary therapy can be an important tool to help you manage EOE. Learn how your food choices can raise or lower your inflammation.
Barrett’s esophagus is an abnormal change in the lining of the esophagus, which is the tube that connects the mouth and stomach.
Scientists have identified a protein that may be the cause of tissue damage in patients with eosinophilic esophagitis (EoE), which affects as many as 56 of every 100,000 people in the United...
Eosinophilic esophagitis, or EoE, is a chronic, immune-mediated condition where your esophagus develops inflammation. Let's cover EoE basics!
Eosinophilic esophagitis or inflammation of the esophagus is sensitive to sugar and may be linked to genetic or acquired thiamine deficiency.
Esophageal ulcers are a type of peptic ulcer that develops between the throat and the stomach. Symptoms include pain or a burning sensation behind or below the sternum, the flat bone that runs down the center of the chest. Using medications, bodily infections, and exposure to stomach acid are the most common causes.
A novel exercise is described for resistance training of the lower esophageal sphincter. Resistance is provided by gravity as food is swallowed and pushed up an incline into the stomach. The incline is established by kneeling with the head bowed lower than the stomach. After several months of daily repetitions, symptoms of gastroesophageal reflux ceased and the exercise was discontinued without relapse.
A mom explains her son's rare disease.
Living with daily struggles and raising awareness for all those suffering from EoE, Eosinophilic Esophagitis and other eosinophil-associated diseases.
Heartburn produce great discomfort. Learn about these home remedies for acid reflux relief and stop that painful body reaction.
Tracheoesophageal Fistulas and Tracheal Anomalies Tracheoesophageal fistula (TOF) and esophageal atresia rarely occur as separate entities, but they are often seen in various combinations: esophageal atresia with upper fistula, lower fistula, and double fistulas. Approximately 10% of infants with esophageal atresia do not have a fistula, but there is a long gap between the esophageal segments. An isolated tracheoesophageal fistula (H or N fistula) can occur without an esophageal atresia. The cause of these congenital anomalies is not well understood. Esophageal atresia is usually sporadic and rarely familial. Maternal polyhydramnios and a small or absent fetal stomach bubble on antenatal ultrasonography suggest the possibility of esophageal atresia antenatally. Postnatally, the diagnosis can be suspected in a newborn infant who has excessive mucus and cannot handle his or her secretions adequately. Suction provides temporary relief, but the secretions continue to accumulate and overflow, resulting in aspiration and respiratory distress. Feeds are also regurgitated and aspirated. The TOF provides a low-resistance pathway for respiratory gases and gastric distension, and subsequent rupture may further compromise ventilation. Formerly, the diagnosis was made by using a contrast study with barium or Gastrografin (meglumine diatrizoate); however, there is the danger of aspirating these materials into the lungs. The diagnosis can readily be made by passing a fairly large radiopaque plastic catheter through the nose or mouth into the pouch. When the catheter cannot be advanced into the stomach, the catheter should then be taped in place and put on constant gentle suction. This keeps the pouch free of saliva and minimizes the chances of aspiration pneumonitis. On the chest radiograph, it will be noted that the tip of the catheter is usually opposite T2-T3. If the surgeon prefers a contrast study, no more than 0.5 mL of contrast material should be introduced through the catheter, with the child in the upright position. Radiography will show the typical esophageal obstruction, and the contrast material should then be immediately aspirated. Initial management is aimed at keeping the airway free of secretions using a 10-Fr double lumen Replogle tube in the proximal pouch on continuous low pressure suction. The ideal surgical procedure consists of disruption of the fistula and an end-to-end anastomosis of the esophagus. If there is a long gap between the esophageal segments, surgery is delayed to allow the pouches to elongate and hypertrophy over a period of up to 3 months. During this time, the infant is fed through a gastrostomy, and the upper pouch is kept clear of secretions. Anomalies and Strictures of The Trachea Tracheal anomalies are very rare. With stricture of the trachea, there is local obstruction of the passage of air. In the absence of cartilage, the trachea can collapse and therefore obstruct on expiration. With deformity of cartilage, there is obstruction on inspiration and expiration. When abnormal bifurcations are present, the right upper or left upper lobe bronchi (or both) arise independently from the trachea. Clinically, stenosis may be localized or diffuse. The localized form is caused by a web of the respiratory mucosa or by excessive growth of tracheal cartilage. The diffuse form is caused by a congenital absence of elastic fibrous tissue between the cartilage and its rings in the trachea or by an absence of cartilage. Clinically, obstruction of the trachea causes chronic dyspnea; cyanosis, especially on exercise; and repeated attacks of respiratory tract infection. The diagnosis is established by bronchoscopy and by radiography. For localized obstruction, surgery is advisable, either dilatation or excision with end-to-end anastomosis. Resection and anastomosis of the trachea can be carried out, including up to six tracheal rings. For generalized stenosis, only supportive therapy is available.
The inflammation or irritation of the esophagus, a tube that carries food from our mouth to the stomach, is called Esophagitis. This condition can be painful and make it hard for you to swallow the…
Having pain in your jaw or neck? It could be Eagle syndrome. Learn how to recognize and treat this rare condition.
Benign esophageal stricture is a narrowing or tightening of the esophagus. Find more information on the causes, symptoms, and treatment of benign esophageal stricture.
