Overexpression of hTau in glia disrupts LDs and glial cell morphology in response to neuronal ROS. Credit: Nature Neuroscience (2024). DOI: 10.1038/s41593-024-01740-1 A study by researchers at Baylor

NeuroSurge: A powerful, dynamic name suggesting precision, innovation, and progress in neurology and technology. Possible uses: A cutting-edge neurological research institute. A tech startup specializing in brain-computer interfaces. An advanced neurology clinic offering innovative treatments.

Researchers and clinicians have often studied genes that affect the function and development of the brain to explain neurological disorders. While we know

Research is constantly looking for a way to identify and diagnose fibromyalgia. This study indicates that researchers may have found a neurological signature. But, is it enough?

Common and rare neurologic manifestations associated with fibromuscular dysplasia include migraine-type headaches, tinnitus, ischemic stroke, and transient ischemic attack.

Brand associations are the mental connections between a brand and people, places, things, and emotions. Being strategic about them will build brand equity.

According to a case report recently published online in JAMA Neurology, researchers describe a rare incident where a man had developed seizures brought on by completing Sudoku puzzles.

Researchers at Barrow Neurological Institute made a Lego checkerboard and placed white and purple M&Ms on the squares in a way that makes the board appear to bulge. [via]

As the COVID-19 pandemic developed and neurological manifestations were reported, concern grew that SARS-CoV-2 might directly invade neuronal cells. However, research throughout the year to address this concern has revealed a different story with inflammatory processes at its centre.

Psoriatic arthritis increases the risk of developing neurological conditions like seizures and migraines. Learn more about how psoriatic arthritis inflammation affects the brain.

Gene reactivation restored hearing after loss in mice, but the timing of intervention is key.

Could Eating Peppers Protect You From Parkinsons?

Ipsen is looking to deepen its expertise in movement disorders in a research collaboration with Skyhawk Therapeutics to develop small molecule drugs for RNA targets in neurological diseases.

Researchers have discovered a molecular complex crucial for the transport of mitochondria within neurons, offering new insights into preventing neurodegenerative diseases.

From the latest neurology news, treatments and therapies, inspiring patient stories, to expert advice, we're here to help you live your healthiest life every day.

Scabies is a skin condition that has plagued humans for centuries, causing discomfort and irritation. However, amidst the wealth of information available, there are still many misconceptions circulating about scabies. In this blog, we aim to debunk five common myths about scabies to provide you with accurate information on this skin condition. Whether you're seeking to understand how to treat scabies or simply want to separate fact from fiction, read on to uncover the truth. 5 Common Myths about Scabies The following myths are common about scabies: Myth #1: Scabies Only Affects Unhygienic People One of the most pervasive misconceptions about scabies is that it only affects individuals who lack proper hygiene. This couldn't be further from the truth. Scabies are caused by the microscopic mite Sarcoptes scabiei, which can infest anyone, regardless of their cleanliness. Scabies are highly contagious and can spread through close physical contact with an infected person, making it crucial to address the issue promptly. Myth #2: Scabies Is Just an Itchy Rash While itching is a hallmark symptom of scabies, the condition is more than just a bothersome rash. Scabies mites burrow into the skin, laying eggs and causing intense itching, especially at night. However, scabies can also lead to secondary infections, skin inflammation, and even complications if left untreated. It's essential to recognize scabies as a parasitic infestation that requires proper medical attention. Myth #3: Scabies Will Go Away on Its Own Some individuals believe that scabies will resolve itself without treatment. This is a dangerous misconception, as scabies will not disappear without intervention. Scabies can worsen over time if left untreated, leading to more severe symptoms and potential complications. To effectively treat scabies, consult a healthcare professional who can recommend appropriate medications and procedures. Myth #4: You Can't Get Scabies Twice Another common myth is that once you've had scabies, you can't get it again. Unfortunately, this is not true. While your body may develop some immunity after an infestation, it's still possible to contract scabies a second time if you come into contact with an infected person or their belongings. It's essential to take precautions and practice good hygiene to minimize the risk of re-infestation. Myth #5: Over-the-Counter Creams Can Cure Scabies Some people believe that over-the-counter creams and lotions can effectively treat scabies. While these products may provide temporary relief from itching, they are unlikely to eliminate the mites and eggs. To treat scabies effectively, prescription medications such as permethrin or ivermectin are typically required. Seek guidance from a healthcare professional to receive an accurate diagnosis and an appropriate treatment strategy. Conclusion In conclusion, debunking these common misconceptions about scabies is essential for understanding how to treat scabies and prevent its spread. Remember that scabies can affect anyone, is more than just an itchy rash, and won't disappear on its own. It's crucial to seek medical guidance and follow prescribed treatments to ensure a complete recovery. Additionally, practicing good hygiene and taking preventative measures are key to avoiding reinfestation. By dispelling these myths, we can better protect ourselves and our loved ones from this contagious skin condition.

Researchers have identified a new, direct signalling pathway through which motor neurons influence locomotion circuits that generate rhythmic movement.

Two professors argue for the importance of neurological research in design

Press release October 1, 2015: A chemical in the brain could potentially be harnessed to help young people with Tourette Syndrome (TS) to overcome the

A new method diagnosing autism re-analyzes MRI scans via AI to detect the movements of proteins, nutrients and other processes in the brain that may indicate the condition

New research spotlights long COVID’s serious cognitive health repercussions, including a 6-point drop in IQ.

This product includes: 1. Editable Vector .AI file Compatibility: Adobe Illustrator CC 2. Editable Vector .EPS-10 file Compatibility: Most Vector Editing Software 3. High-resolution JPG image 6000 x 3140 px License terms in short: Use for everything except reselling item itself. Read a full license here

Like so many new pathways to health, promising new treatments for rheumatoid arthritis, depression, epilepsy, and more got their start as a surprise. A research team studying the vagus nerve—which links all of our major organs to the brain—found that the nerve had an unexpected ability to prevent inflammation, in unexpected places. Hello, vagus nerve, pleased to meet you If you don’t have the vaguest idea about the vagus nerve, I think you’ll be impressed. It runs from your brainstem down to your abdomen, passing through the heart, esophagus, stomach, and lungs along the way. As part of our involuntary nervous system, it controls what our bodies do unconsciously—regulating our heart rate, managing the digestive processes, and keeping us breathing, for example. Our new understanding of how it works is the result of an inspiring story about surprises in the research…followed by educated guesses, ingenious experiments, stubborn persistence, and finally, success. And talk about dedication—we’re talking about more than 10 years of hard work. A shout out to all you determined researchers out there. We owe you. In the beginning… Looking for ways to reduce stroke-related inflammation in the brain, the research team injected a small amount of an anti-inflammatory drug into a lab animal’s inflamed brain. They noticed that the drug didn’t just block inflammation in the animal’s brain—it also blocked inflammation in its spleen. This was totally unexpected. The amount of the drug in the brain was far too small to have entered the bloodstream and traveled to other parts of the body. Clearly, there was another channel of communication between the brain and the spleen. Hmmm… Why? How? Good science needs good hypotheses. Maybe the brain was using a particular part of the nervous system—the vagus nerve—to instruct the spleen to switch off inflammation. That is, perhaps the brain was directly regulating inflammation in body tissues. Well…except for one thing. That hypothesis slammed headlong into what we thought we knew: Communication between the immune system cells in our bloodstream and organs, using the electrical connections of the nervous system, was considered impossible. Indeed, the brain has an independent, autonomous nervous system of its own, protected from unwanted outside influence by the blood-brain barrier. Even so, here, it looked like the two systems were intimately, intricately linked. Time to test the hypothesis by cutting the vagus nerve, injecting the same anti-inflammatory drug into the same part of an identical lab animal’s brain. The result? With the vagus nerve disconnected, the anti-inflammatory drug had no effect elsewhere in the body. OK, so now we test whether we can stimulate a healthy vagus nerve to send out anti-inflammatory instructions—but using electrical signals that we generate, instead of the anti-inflammatory drug. Result? Same as with the drug. Healthy vagus nerve plus electrical stimulation blocked inflammation in the spleen. Breakthrough! The vagus nerve helps the brain communicate with more of our body than anyone knew. Through the newly-opened door The research team spent the next ten years mapping every neural pathway along the length of the vagus nerve. They finally nailed how the vagus nerve controls inflammation. It sends a message to the spleen to cease production of an inflammation-fueling protein called, tumor necrosis factor (TNF). So it was time for the real test. A test of nerve In 2011, volunteers suffering severe rheumatoid arthritis, and taking conventional medications to suppress its symptoms, were implanted with a small pacemaker-like device with wires attaching to the vagus nerve in their throat. They were instructed to swipe a magnet across their throat six times a day. This would, they hoped, activate the implant to stimulate the vagus nerve for 30 seconds at a time, which would in turn reduce the inflammatory response caused by their arthritis. The hope was for a modest reduction in pain. The results far exceeded that. A few weeks after the test, a 68-year old grandmother of eight, who was often virtually immobilized by her arthritis, was back playing senior volleyball, bicycling for an hour a day, and doing gymnastics. She will need to continue a low dose of her prescription drug indefinitely, but finds that no obstacle. Others had similarly life-changing experiences: Of the 20 patients in the trial, 16 not only reported they felt better, but also had reduced measures of inflammation in their blood More than half showed significant improvement, and around one-third are in full remission—fully cured, in effect, of their rheumatoid arthritis, for which there had never been a cure Even those few who experienced no clinically significant improvements with the implant insisted it helped them. Nobody wanted theirs removed. How happy an ending is this? Follow-up studies by the research team underscored the effectiveness of vagus nerve stimulation: When stimulation was temporarily discontinued, the arthritic pain returned, and TNF levels in the blood increased When stimulation was restarted, symptoms again disappeared I call that a very well done deal. A happy ending that marks an equally happy beginning, as brilliant researchers continue to work out how vagal stimulation can improve the health and lives of millions of people. What’s ahead? This happy ending gets even happier. On the heels of vagal stimulation successes with rheumatoid arthritis, came advances in the treatment of epilepsy and severe depression. And an implantable device is under study for the treatment of heart failure. And new, noninvasive vagus nerve stimulation devices, requiring no implantation, have been approved in Europe to treat epilepsy, depression, and pain, but have not yet been approved in the U.S. Others are currently studying vagus nerve stimulation as a potential treatment for multiple sclerosis, headache, pain, and Alzheimer’s disease. And there are even some health care practitioners using manual vagus nerve stimulation. That research team and all those who have followed have truly struck a nerve. If you’re epileptic or severely depressed, vagus nerve stimulation might be a life-changing help. Make sure your doctor knows about it. Take good care. References Gaia, Vince. “Hacking the nervous system” Published May 26, 2015. Last accessed January 22, 2017. “Vagus Nerve Stimulation” Mayo Clinic. Published December 24, 2015. Last accessed January 22, 2017. “Vagus Nerve” Healthline Medical. Reviewed April 15, 2015. Last accessed January 22, 2017. Tewfik,Ted. “Vagus Nerve Anatomy” Updated Aug 17, 2015. Last accessed January 22, 2017. Rosenfeld, Jordan. “9 Nervy Facts About the Vagus Nerve” Mental Floss. Published July 2, 2015. Last accessed January 22, 2017. Last Updated: August 16, 2018Originally Published: February 15, 2017

There are multiple scales that are commonly used worldwide in the assessment of stroke victims. Learn about the 5 most common scales at CareerCert.

“We found that individuals exposed to cats had approximately twice the odds of developing schizophrenia,” researchers recently revealed.